Biology 457 & 557 -  Week 6

 

General mechanisms which control transcription

1) primary DNA modification correlated with genetic regulation is methylation

            w in mammals, -CH3 groups added to

           

            w gene inactivation by methylation of cytosines places methyl groups into the

           

           

            w also possible that certain regulatory proteins recognize and bind methylated

               DNA

            w inactive genes are highly methylated in CG islands

           

            w 1 of 2 X chromosomes in female mammalian somatic (body) cells is almost

               completely inactivated

            inactive heterochromatin

 

            w methylation patterns of globin genes best studied vertebrate model

                       

                       

                       

 

 

 

 

 

 

 

2) chemical modifications of histones by acetylation and phosphorylation

 

            w histones organize DNA into nucleosomes

                        - octamer of H2A, H2B, H3, H4 wrapped with ~165 bp DNA (~2 turns)

                        - H1 binds in region where DNA enters/exits the nucleosome

                          (stabilizes DNA)

                                           O

                                          

            w acetylation (add -C-CH3) of lysine residues by acetyltransferase

                        - this is reversible by

            w core histones are acetylated in regions that interact with DNA 

           

 

 

 

 

            w nucleosomes unfold and regulatory proteins/transcription factors

           

            w may be complete unfolding/partial unfolding

            w recently found that a component of the basal transcription factor TFIID

           

            w phosphorylation of H1 converts chromatin into inactive heterochromatin

                        - induces packing association

                        - attraction of H1 molecules

                        - dephosphorylation

 

 

 

 

 

 

 

CONCLUSIONS:

ä THESE CONTROL MECHANISMS I HAVE DESCRIBED ACT IN CONCERT TO

    TURN GENES ON AND OFF AT THE PROPER TIMES AND IN THE PROPER

    PLACES USING THE CORRECT COMBINATION OF REGULATORY

    ELEMENTS AND SEQUENCES

 

 

 

 

 

 

 

 

ä CONTROLLING GENE ACTIVITY PROPERLY IS IMPORTANT FOR

    EMBRYONIC DEVELOPMENT THROUGH MAINTENANCE OF ADULT

    ACTIVITIES AND IS A VERY COMPLEX PROCESS

 

 

 

 

 

 

 

 

 

 

 

Transcriptional Regulation in Prokaryotes

 

            because bacteria normally live in a constantly changing environment, most of

their regulatory controls are easily reversible systems

 

 

very few bacterial regulatory pathways lead to permanent cellular changes

 

 

bacterial genes are organized in operons…..

 

each operon is controlled as a unit by one or more promoters

 

 

the 5’ flanking regions of bacterial operons are recognized and bound by

regulatory proteins called

 

            repressors adjust the rate of initiation

           

            repressor-regulated operons are characterized by increased transcription

             

 

 

activators work in an opposite fashion and adjust their promoters upward

from a low base level

 

 

 

operons are also regulated by sigma factors

 

            these factors have the same overall effect as activators

           

           

 

many operons are controlled by more than one regulatory mechanism

 

 

 

            the result is a complex network of superimposed control

           

           

 

 

best studied example of this is the lac operon

 

            there are 3 genes in this operon (transcriptional unit) coding for 3 enzymes

involved in lactose metabolism

 

 

 

 

 

 

 

 

 

this lac operon is controlled by a regulatory protein called the lac repressor

            this repressor is encoded by a gene separate from the lac operon

 

 

            in an active form, the repressor binds to a site within the promoter

 

 

 

 

 

 

 

 

 

 

when the repressor is bound to the operator, RNA polymerase can’t bind

tightly

 

so the repressor binds when no lactose is present

 

            the lac repressor also has a binding site

           

 

            this binding induces a conformational change in the repressor

           

 

            RNA polymerase can now bind and transcribe the operon

 

 

 

 

 

            this type of repressor system ensures that the enzymes of the pathway are not

            made unless they are required

           

 

 

the repressor-based mechanism can also reduce the synthesis of the enzymes of a

pathway when a product of the pathway is present in the medium

 

            ex: trp operon,

           

 

            this system also has a repressor, trp repressor, which has 2 binding sites, one

            for the promoter of the trp operon and the other one for tryptophan

 

 

            however, unlike the lac operon repressor,

 

 

            in this form, the repressor has no affinity for the trp promoter

           

 

            this is the situation when the supply of tryptophan is low

 

 

 

 

 

 

 

 

 

            if tryptophan becomes available or if cellular levels of the amino acid are high

            because of synthesis by this pathway, then excess tryptophan can bind to the

            repressor

 

 

 

 

 

 

 

 

 

 

 

            thus, when tryptophan is available the pathway is off

 

 

 

a few more details of repressors:

            in the case of the lac repressor, its 3-D structure has been determined

 

 

            the tetramer contains helix-turn-helix motifs

 

 

            most other repressors are homodimers

            which contain alpha helices

 

 

            the genes coding for repressors are also operons

 

 

            the operons encoding most repressors are autoregulated

 

 

            as the quantity of repressor molecules increases

 

            autoregulation ensures that there are always some repressor molecules

 

 

 

mention a few words on activators:

 

            typically, activators control groups of operons

 

 

            activators are also autoregulated

 

            have found positive regulators of the lac and 2 other operons, the gal and ara

            operons

 

        ex: cAMP receptor protein (CRP) or catabolite activator protein (CAP)

 

            CRP is activated by cyclic AMP

 

 

            CRP (CAP) is made in inactive form

 

 

            when glucose concentration is low

 

 

            combination with cAMP induces a conformational change

 

            this active cAMP-CRP (CAP) molecule, in the presence of RNA polymerase, can

then bind to the operators of the lac, gal and ara operons simultaneously

 

 

 

            activation of these operons allows the production of enzymes which can

metabolize these other sugars

 

 

if glucose then becomes available again, the enzyme converting ATP to cAMP is

inactivated